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A genetic marker linked to a type of hair loss was found in most patients studied.

Certain genetic variants are linked to frontal fibrosing alopecia in Spanish patients.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

Frontal Fibrosing Alopecia's cause is unclear, affects mainly postmenopausal women, and current treatments focus on stopping hair loss rather than regrowth.

Graft-versus-host disease is a complication where donor immune cells attack the recipient's body, often affecting the skin, liver, and gastrointestinal tract.

Hormonal imbalances during menopause may significantly contribute to Frontal Fibrosing Alopecia.

The article suggests that patients with Frontal Fibrosing Alopecia may have more contact allergies, but it doesn't prove that allergies cause the condition.

Frontal Fibrosing Alopecia is a hair loss condition mainly affecting postmenopausal women, with unclear causes and various clinical patterns.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Oral contraceptive use may increase the risk of frontal fibrosing alopecia in women with a specific CYP1B1 gene variant.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Certain genetic variants linked to immune response increase the risk of alopecia areata in Taiwanese people.

Certain HLA class II alleles increase or decrease the risk of alopecia areata.

Hair loss from Alopecia Areata is caused by both genes and environment, with several treatments available but challenges in cost and relapse remain.

A severe medication reaction required long treatment and led to hair loss and thyroid issues.

Alopecia areata has a complex genetic basis that was not fully understood as of 2001.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

Thymus transplantation successfully restored immune function in infants with FOXN1 deficiency.

People with alopecia areata have higher levels of osteopontin, which might be important in the disease's development, but this doesn't relate to how severe the disease is.

Asian patients with Frontal Fibrosing Alopecia often lose eyebrow hair and respond well to combined antiandrogen or antimalarial and topical treatments.

The document concludes that various findings in rheumatology offer insights into disease severity, treatment responses, and potential risks in medication, with some limitations due to unspecified participant numbers.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Certain genetic markers, especially the MICA gene, are linked to alopecia areata.

Certain KIR genes in Indian SLE patients are linked to disease severity and could be biomarkers.

Genetic variants linked to ten skin diseases were found, showing both immune and non-immune factors play a role.

Protein profiling of forehead skin can help distinguish between frontal fibrosing alopecia and androgenetic alopecia.

Familial frontal fibrosing alopecia is rare, mostly affects women, and often occurs between sisters or mother-daughter pairs.

COVID-19 vaccination does not significantly increase the risk of developing alopecia areata.

The report concludes that PCOS is mainly a condition of excess male hormones and its definition may change as new information is discovered.