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Men with baldness due to androgenetic alopecia still have hair stem cells, but lack specific cells needed for hair growth.

Activating the Sonic hedgehog gene in mice can start the hair growth phase.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

Nanoparticles can deliver vaccines through hair follicles, triggering immune responses and providing protection.

Skin-derived precursors in hair follicles come from different origins but function similarly.

Wharton’s Jelly stem cells from the umbilical cord improve skin healing and hair growth without scarring.

Prolactin affects hair growth and skin conditions, and could be a target for new skin disease treatments.

BMP signaling controls hair follicle size and cell growth by affecting cell cycle genes.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Drosha and Dicer are essential for hair follicle health and preventing DNA damage in skin cells.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Fat cells are important for healthy skin, hair growth, and healing, and changes in these cells can affect skin conditions and aging.

The Hedgehog signaling pathway is important for skin and hair growth and can lead to cancer if it doesn't work right.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

PRP injections may be a safe, effective alternative for hair loss treatment compared to minoxidil and finasteride.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

Spermidine may help reduce hair loss and deserves further testing as a treatment.

Most hair follicle stem cells do not protect their DNA by dividing it unevenly.

Fat from the body can help improve hair growth and scars when used in skin treatments.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

Special cell particles from macrophages can help hair grow.

Pitx2 helps outer root sheath cells differentiate but can't start hair growth on its own.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Gray hair may be caused by lower antioxidant activity in hair cells.

The Wnt/β-catenin pathway can activate melanocyte stem cells and may help regenerate hair follicles.

There are two types of stem cells in rodent hair follicles, each with different keratin proteins.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

3,4,5-tri-O-caffeoylquinic acid promotes hair growth by activating the β-catenin pathway.