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Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Two-photon microscopy effectively tracks live stem cell activity in mouse skin with minimal harm and clear images.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

FoxN1 overexpression in young mice harms immune cell and skin development.

Fat stem cells from diabetic mice can help heal skin wounds in other diabetic mice.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

Dermal adipose tissue in mice can change and revert to help with skin health.

Activating β-catenin in certain skin cells speeds up hair growth in mice.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Hair follicle stem cells can become motor neurons and reduce muscle loss after nerve injury.

Implanted special stem cells from hair follicles helped heal wounds faster and with less scarring in mice.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Autophagy prevents early aging and maintains lipid and pheromone balance in mouse glands.

A mouse gene mutation increases the risk of skin cancer.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

A substance called FGF9 from certain immune cells can trigger new hair growth during wound healing in mice, but humans may not have the same response due to fewer of these cells.

Regulatory T cells are essential for normal and improved wound healing in mice.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Tissue stiffness affects hair follicle regeneration, and Twist1 is a key regulator.

FGF9 from certain T cells helps create new hair follicles during wound healing, which could potentially be used for hair loss treatments.

Decorin helps hair cells grow and move, and keeps hair growth phase going in mice.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Loss of Msx2 function causes eye development issues similar to Peters anomaly.