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Using radiation to make mice's hair turn gray helps study and find ways to prevent or reverse hair graying.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Rosemary oil can boost hair growth by increasing certain enzyme activities and cytokine expression.

Polygoni multiflori Radix water extracts promote hair growth in mice.

Certain enzymes and a growth factor were higher in mice treated with minoxidil or a peppermint and rosemary oil mix, which may be good indicators of hair growth.

Immune changes and specific genes contribute to male hair loss.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

PPARγ is crucial for skin health but can have both beneficial and harmful effects.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Substance P may worsen acne by increasing inflammation, but corticosteroids might help by reducing this effect.

New understanding of the causes of primary cicatricial alopecia has led to better diagnosis and potential new treatments.

PPAR-γ helps control skin oil glands and inflammation, and its disruption can cause hair loss diseases.

GMG-43AC may help reduce unwanted hair growth and treat certain hair loss conditions.

Somatostatin may help protect hair follicles from immune attacks.

PPAR-γ is important for healthy hair and its problems, and more research on PPAR-γ treatments is needed.

Apremilast did not work for treating severe alopecia areata.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Astragalus membranaceus helps regrow hair and reduce hair loss symptoms.

Sorafenib may cause hair loss in a way similar to alopecia areata.

The symposium concluded that a specific lipid might cause hair loss, inflammation is a key factor, and stem cells in bald scalps aren't working right, suggesting new treatment targets.

Chronic inflammatory skin diseases are caused by disrupted interactions between skin cells and immune cells.