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The research found that the molecule lncRNA-H19 helps hair follicle cells grow by affecting certain cell pathways in cashmere goats.

Researchers found a way to grow cashmere goat hair cells in a lab and discovered that certain conditions improve these cells' growth and characteristics.

Genes related to calcium signaling and lipid metabolism are important for curly hair in Mangalitza pigs.

Toothpaste containing α-tocopherol acetate, l-menthol, and stevioside can promote hair growth in mice.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Msx2 and Foxn1 are both crucial for hair growth and health.

The Foxn1 gene is essential for normal nail and hair development.

FOXN1 gene variants cause low T cells and immune issues from birth.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

New treatments for immune disorders caused by FOXN1 deficiency are promising.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Foxn1 helps skin cells mature by controlling a specific protein's activity.

FOXN1 mutations cause severe immunodeficiency, hair loss, nail issues, and thymus defects.

FoxN1 gene is essential for proper thymus structure and preventing hair loss.

A rare genetic mutation found in an Indian family can be detected through prenatal screening.

FoxN1 overexpression in young mice harms immune cell and skin development.

Removing a specific gene in certain skin cells causes hair loss in mice by disrupting hair follicle development.

Removing HIF-P4H-2 from certain skin cells in mice causes hair loss on the body but not the head.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

The gene Foxn1 is important for hair growth, and understanding it may lead to new alopecia treatments.

Early over-expression of FoxN1 harms immune and skin development.

FoxN1 gene is crucial for proper thymus structure and normal skin appearance.

FOXC1 boosts SFRP1 in hair loss, suggesting new treatments.

Thymus transplantation successfully restored immune function in infants with FOXN1 deficiency.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.