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Finasteride may help treat Neutrophil Actin Dysfunction by reducing LSP1 gene activity.

Aging changes skin cells, leading to different DNA methylation and gene activity, affecting cell metabolism and aging signs.

The study found that p63 needs signals from morphogens to help skin cells differentiate properly.

Short-chain fatty acids from *Cutibacterium acnes* cause skin inflammation, contributing to acne.

Elastic Net DNA methylation clocks are inaccurate for predicting age and health status; a "noise barometer" may better indicate aging and disease.

Understanding and manipulating epigenetic changes can potentially lead to human organ regeneration therapies, but more research is needed to improve these methods and minimize risks.

Epigenetic changes contribute to autoimmune skin diseases.

Epigenetic mechanisms control skin development by regulating gene expression.

Genes and epigenetic changes are important in the development of Polycystic Ovary Syndrome.

Colorectal cancer development involves both genetic changes and epigenetic alterations like DNA methylation and microRNA changes.

A mother's diet at conception can cause lasting genetic changes in her child.

Increasing mir-302 turns human hair cells into stem cells by changing gene regulation and demethylation.

The environment around the time of conception can change the VTRNA2-1 gene in a way that lasts for years and may affect disease risk.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

Minoxidil and Finasteride are effective for male baldness; more research is needed for hair aging treatments.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

Androgens are important for normal ovarian function and estrogen production, but may not be the main cause of follicle death.

Researchers found 71 genetic regions linked to male pattern baldness, which account for 38% of its genetic risk.

Chromosomal changes, including those in the WRN gene and rDNA, may significantly contribute to aging.

High levels of androgens during early development may cause PCOS-like symptoms.

The document concludes that understanding hair follicle cell cycles is crucial for hair growth and alopecia research, and recommends specific techniques and future research directions.

Finasteride can cause serious side effects in some men, needing more research for treatment.

Gonadal hormones significantly affect the severity of alopecia areata in mice.

DNA methylation changes are linked to hair growth cycles in goats.

New gene identification techniques have improved the understanding and classification of inherited hair disorders.

Skin aging is caused by stem cell damage and can potentially be delayed with treatments like antioxidants and stem cell therapy.

Androgenetic alopecia is mainly caused by genetic factors and increased androgen activity, leading to hair follicle miniaturization.

Genetic research has advanced our understanding of skin diseases, but complex conditions require an integrative approach for deeper insight.

The methylation of the HOXC8 gene's exon 1 affects cashmere fiber length in goats.