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Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Caspase-14 is important for skin and hair development in all mammals.

Excess vitamin A changes skin and hair follicle development and affects polysaccharide distribution.

A vitamin D3 compound can reduce skin cell growth.

The enzyme type 1 5α-reductase is more active in the hair follicle's lower part than in the skin's outer layer.

Dermoscopy can non-invasively detect eruptive vellus hair cysts on the labia majora.

COX-2 levels change during the hair cycle and affect skin and hair growth.

Claudin expression changes help the skin respond to injury.

CD99 is highly present in certain skin cells and could help treat skin conditions.

These methods help understand DNA changes in mouse skin.

VEGFR-2 is active in hair follicles, sebaceous glands, sweat glands, and skin on the human scalp.

A more sensitive assay was developed to detect enzyme activity converting arginine to citrulline in hair follicles.

The skin and mucous membranes can regenerate over the basement membrane after damage, using nearby surviving cells.

Wnt4 protein makes the outer skin layer thicker in burn wounds by turning on a specific healing pathway and loosening the connections between skin cells.

Artificial dermal template treatment can stimulate complete skin and hair follicle regrowth.

Claudin-1 is crucial for maintaining skin barrier and preventing inflammation.

Epidermis and dermis cells together can regenerate hair follicles.

CTCF protein is essential for skin and hair follicle development in mice.

The created skin model with melanoblasts improves the study of skin color and offers an alternative to animal testing.

The epidermis is crucial for hair growth.

One type of progenitor cell can maintain normal skin in mice.

All types of alopecia involve hair follicle damage, especially in alopecia areata.

α3β1-integrin is crucial for maintaining normal hair follicle shape and function but not needed for the development of the surrounding skin.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

The upper dermal sheath can regenerate hair in rats.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Connexin 30 is usually absent in normal skin but can appear in certain skin conditions.

Keratinocytes from dog hair follicles can create a functional skin layer in a lab model, useful for dog skin therapy.

Different genes are active in dogs' hair growth and skin, similar to humans, which helps understand dog skin and hair diseases and can relate to human conditions.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.