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Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

DHT reduces a cell's ability to promote hair growth, while 3D culture without DHT improves it.

Cerasus serrulata flower extracts have strong antibacterial and antioxidant effects and may help prevent hair loss.

Leonuri Herba alkaloids may help treat hormone-induced enlarged prostate in rats.

Steroid sex hormones activate matriptase in prostate cancer cells but not in breast cancer cells.

Finasteride effectively treats hirsutism in women, but more research needed for long-term results.

Male pattern baldness involves hormone-related hair thinning, shorter hair, and inflammation.

Dihydrotestosterone treatment can help penis growth in boys with 5α-reductase deficiency but doesn't fully normalize size after puberty.

Astragalus sinicus extracts may help promote hair growth and treat hair loss.

Nanoparticles may improve caffeine delivery for hair growth, offering a potential alternative to minoxidil for hair loss treatment.

The inhibitor DPP can promote hair growth.

NMN could potentially treat hair loss by reducing oxidative stress and improving cell health.

Endurance exercise may help BPH by lowering hormones and inflammation.

Minoxidil and finasteride are effective for male hair loss; minoxidil also helps female hair loss, with some treatments needing more research.

A new treatment for hair loss shows promise with fewer side effects.

Combining Finasteride and Minoxidil effectively treats hair loss in middle-aged men.

AGA is caused by genetics and androgens, treatable with finasteride and minoxidil.

Finasteride can have lasting negative effects on brain function and behavior by disrupting neurosteroid production.

Higher enzyme levels found in women with hair loss, suggesting hormone imbalance.

Post-finasteride patients show changed neuroactive steroid levels, possibly causing erectile dysfunction and depression.

Finasteride treatment lowers urine androgen and prostaglandin levels, making them potential markers for effectiveness.

Finasteride improves acne and hair loss in women with normal testosterone.

Male hormones, particularly DHT, are linked to male pattern hair loss, and treatments like finasteride can help, but they don't work for postmenopausal women's hair loss, which may have different causes.

Women with low SHBG levels and a high 3a-diol G to SHBG ratio are likely to experience female pattern baldness, possibly due to a slight excess of androgens affecting sensitive hair bulbs.

DHT's role in hair loss is important, but measuring its level for diagnosis is questionable.

A drug improved schizophrenia-like symptoms in stressed rats by changing brain steroid levels.

Hair transplantation is a treatment for hair loss mainly caused by genetics, with various techniques and potential complications, and results visible after 8-12 months.

Genes and hormones cause hair loss, with four genes contributing equally.

Finasteride cream reduces hair growth in women with hirsutism, but more research needed.

The developed model can predict effective 5-alpha-reductase enzyme inhibitors.