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Hair follicle-derived extracellular vesicles may help heal chronic wounds as effectively as those from adipose tissue.

Misbehaving hair follicle stem cells can cause hair loss and offer new treatment options.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Ruxolitinib helps protect skin stem cells and keeps skin healthy in mice with skin GVHD.

Antigen presenting cells around hair follicles are crucial in SLE-related hair loss.

Choosing the right method to separate skin layers is key for good skin cell research.

Immune cells are essential for early hair and skin development and healing.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Alopecia areata, a type of hair loss, may be passed through T cells and has genetic links, while treatments vary in effectiveness. Male pattern baldness can be treated with finasteride and is influenced by androgens in hair follicles.

Glycerol may improve wound healing and prevent keloids, a device can measure itch intensity, male pattern baldness is highly heritable, and fumaric acid esters may work for psoriasis by causing cell death in T cells.

ADSCs help in wound healing and skin regeneration but need more research for full understanding.

Bacteria can help skin regenerate through a process called IL-1β signaling.

Older mice healed wounds better but lost more weight and might have weaker immune systems afterward.

Skin structure complexity and variability are crucial for assessing skin toxicity in safety tests.

Oxidative stress contributes to hair graying and loss as we age.

IL-2 treatment causes skin eruptions and other reversible side effects, and may play a role in psoriasis.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Follicular Cell Implantation might become a new treatment for hair loss and could lead to advances in organ regeneration.

Quercetin significantly helps hair growth by activating hair follicles and improving blood vessel formation around them.

CXXC5 is a protein that prevents hair growth and could be a target for hair loss treatment.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

Lymphatic vessels help hair follicles regenerate by interacting with stem cells.