Search

everythinglearnresearchcommunity

for

Search:↓

Researchers developed a way to study human body clocks using hair tissue, which works similarly in both healthy and dementia patients.

Hair grows faster in the morning and is more vulnerable to damage from radiation due to the internal clock in hair follicle cells.

A gene called BMAL1 plays a role in controlling hair growth.

The circadian clock is crucial for tissue renewal and regeneration, affecting stem cell functions and having implications for health and disease.

Night shift work disrupts the body's natural clock genes.

The circadian clock influences the behavior and regeneration of stem cells in the body.

Understanding and tracking our body's natural daily rhythms could help improve heart health.

Hair follicles could be used to noninvasively monitor our body's internal clock and help identify risks for related diseases.

BMAL1 controls skin cell growth and UV damage risk, peaking at night.

Circadian rhythms are crucial for stem cell function and tissue repair, and understanding them may improve aging and regeneration treatments.

Photobiomodulation therapy using red and near-infrared light can reduce inflammation and aid in healing various conditions.

Children with atopic dermatitis often have sleep problems, affecting their growth and behavior.

Certain genes control the color of human hair by affecting pigment production.

Different light affects cell functions and can help treat skin conditions.

Circadian clock genes are important for hair growth and may affect aging-related hair loss and graying.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Natural skincare products may help reduce sun damage and support the skin's daily cycle.

The circadian clock plays a key role in hair growth and its disruption can affect hair regeneration.

Different types of cell death affect skin health and inflammation, and understanding them could improve treatments for skin diseases.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

ZDHHC13 is important for normal liver function and metabolism, affecting mitochondrial activity.

A mother's PPARγ is crucial for preventing harmful milk that can cause inflammation and growth problems in babies.

Caspases are enzymes important for both cell death and various non-lethal cell functions, affecting head development and hair growth, with different caspases playing specific roles.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

FA2H is essential for normal fur and sebum production in mice.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Rapamycin may help treat Leigh syndrome by targeting protein kinase C.