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Minoxidil is more effective than Latanoprost for treating patchy alopecia areata.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

Baricitinib treatment helped reduce hair loss symptoms in mice by decreasing inflammation-related immune cells.

Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

Phosphodiesterase inhibitors like tadalafil can reduce cell growth in BPH caused by CD8+ T cells in low androgen conditions.

A rare type of skin cancer with mucosal involvement was partially treated but eventually relapsed.

Biopsy can differentiate between alopecia areata and androgenic alopecia, and if more information is needed, testing for CD3 and CD8 can help.

Assessing CD8+ T cell levels before surgery may improve vitiligo treatment outcomes.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Alopecia Areata is an autoimmune condition causing hair loss with no cure and treatments that often don't work well.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Alopecia areata in these mice is inherited, more common in young females, and can be treated with triamcinolone acetonide.

Alopecia areata is caused by the immune system attacking hair follicles.

Alopecia areata is an autoimmune disease causing hair loss, treatable with immune-modulating drugs, and linked to genetics.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Alopecia areata involves immune system imbalances that may lead to depression and anxiety.

Apremilast may help treat hair loss in alopecia areata.

JAK inhibitors might help treat alopecia areata.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Frontal fibrosing alopecia is linked to increased immune system activity and reduced stem cells, suggesting early treatment targeting this pathway might prevent hair follicle damage.

Alopecia Areata might be linked to Crohn's disease.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Serum granulysin levels can indicate the activity and prognosis of alopecia areata.

Frontal Fibrosing Alopecia's cause is unclear, affects mainly postmenopausal women, and current treatments focus on stopping hair loss rather than regrowth.

People with alopecia areata have fewer regulatory T-cells than those with other skin conditions.

Ginsenoside Rg3 may help hair growth by increasing a growth-related protein in hair cells.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.