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Fibrosis in the bulge area of hair follicles can cause hair thinning in male pattern baldness, and drugs that inhibit fibrosis might help reverse this.

Hair follicles help attract immune cells to the skin during stress.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

A new method using Y-27632 improves the growth and quality of human hair follicle stem cells for tissue engineering and therapy.

The new method using gene-modified stem cells and a 3D printed scaffold improved skin repair in mice.

Adipose-derived stem cells may help with hair loss, but more research is needed.

PRP shows promise for treating mild alopecia areata but needs more research for cicatricial alopecias.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Alopecia areata severity and treatment response are linked to specific cytokine levels.

Blocking a protein called CXXC5 with a specific peptide can stimulate hair regrowth and new hair growth in wounds.

Ifidancitinib, a JAK inhibitor, effectively regrows hair in mice with alopecia by tiring out harmful T cells.

The protein EZH2 blocks microRNA-22, increasing STK40 protein, which helps hair follicle stem cells change and grow hair.

The study identified 12 potential biomarkers for hair loss and how they affect hair growth.

Different genes are active in dogs' hair growth and skin, similar to humans, which helps understand dog skin and hair diseases and can relate to human conditions.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

Minoxidil nanoparticles significantly boost hair growth in mice compared to regular minoxidil.

The LncRNA AC010789.1 slows down hair loss by promoting hair follicle growth and interacting with miR-21 and the Wnt/β-catenin pathway.

Trichoscopy helps tell apart Frontal Fibrosing Alopecia and Lichen planopilaris by showing different hair and scalp features.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Autophagy helps activate hair stem cells and hair growth by changing their energy use to glycolysis.

Stem cell therapies show promise for hair regrowth in androgenetic alopecia.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.

Stem cells are crucial for wound healing and understanding their role could lead to new treatments, but more research is needed to answer unresolved questions.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Stress may trigger hair loss by affecting immune protection in hair follicles.

FGF13 gene changes cause excessive hair growth in a rare condition.

Estrogen can temporarily slow down hair growth but this can be reversed.

The document concluded that more research is needed to find the best treatment for Frontal fibrosing alopecia.

PPAR-γ helps control skin oil glands and inflammation, and its disruption can cause hair loss diseases.