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A girl with Crohn's disease developed hair loss due to her increased medication, a rare side effect seen in some children.

Botulinum toxin type A significantly reduces scalp psoriasis severity compared to placebo.

Tph cells are linked to the severity of systemic lupus erythematosus.

NK cells play a role in skin diseases like eczema and psoriasis.

MDSC-Exo can treat autoimmune alopecia areata and promote hair regrowth in mice.

People with cutaneous Lichen Planus are more likely to have Metabolic Syndrome and related conditions like high cholesterol, diabetes, and high blood pressure.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Fat tissue under the skin affects hair growth and aging; reducing its inflammation may help treat hair loss.

Japanese patients with alopecia areata often have a higher BMI and consume more vitamin C, fruit, and retinol, which may affect their condition's development or severity.

Blocking JAK3 signaling can reverse hair loss from alopecia areata.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

Men and women get COVID-19 at similar rates, but men tend to get sicker and have a higher risk of dying, while women usually have stronger immune responses and vaccine reactions.

Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

Oxidative stress plays a significant role in alopecia areata, and new treatments may include JAK inhibitors and antioxidants.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Dihydrotestosterone treatment on 2D and 3D-cultured skin cells slows down hair growth by affecting certain genes and could be a potential target for hair loss treatment.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Alopecia areata and vitiligo share immune system dysfunction but differ in specific immune responses and affected areas.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

CXCL12 protein slows down hair growth through its receptor CXCR4. Blocking this can potentially increase hair growth.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

New targeted immunotherapies are improving treatment for inflammatory skin diseases.

Hair follicle stem cells reduced hair loss and inflammation in mice with a condition similar to human alopecia.

Topical JAK inhibitors can effectively treat alopecia areata and vitiligo by modulating immune responses.

Hair follicles help attract immune cells to the skin during stress.

Keratin proteins are increasingly recognized as important for cell health and are linked to many diseases.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

Alopecia areata is an incurable autoimmune condition causing hair loss, with research aiming for better treatments.

Activating Notch signaling can kill basal cell carcinoma cells.

Blocking Janus kinase 1 helps stop inflammation and regrow hair, making it a good treatment for hair loss from alopecia areata.