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Altered retinoid metabolism in cicatricial alopecia suggests a balanced vitamin A diet may prevent the condition.

High-dose vitamin A and E with zinc improved blood sugar control and insulin function in diabetics, but may cause hair loss.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Low levels of vitamin B12 and ferritin are linked to early hair graying.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

A vitamin D receptor mutation causes rickets and affects immune responses.

Men and premenopausal women in Korea show different patterns in iron and vitamin D levels, with no clear pattern for postmenopausal women.

Both topical calcipotriol and narrowband UVB improved alopecia, but combining them didn't enhance the effect on hair loss severity, despite higher vitamin D3 levels.

Vitamin D might help treat some types of hair loss, but more research is needed.

Telogen effluvium is linked to deficiencies in iron, vitamin B12, and thyroid function.

Low vitamin D3 and ferritin levels may cause hair loss in pregnant women.

A rare genetic disease causes rickets and often hair loss in young children, which can improve with specific treatments.

Two siblings with a rare type of rickets and hair loss had low vitamin D levels, which is not typical for their condition.

Low levels of vitamin B12, ferritin, and calcium are linked to premature graying of hair.

Skin biopsies are recommended for confirming alopecia diagnosis due to variability in clinical assessments; the link between vitamin D levels and alopecia is unclear.

Combining microneedling with either vitamin D3 or bimatoprost improves hair regrowth more than microneedling alone.

Low vitamin D levels can significantly contribute to hair loss, especially in women aged 35-45. Correcting these levels early may help prevent and treat this condition.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

European guidelines recommend regular eye and ear exams, skin care, vitamin D supplements, and cautious use of medications for managing congenital ichthyoses.

The vitamin D receptor can work without its usual activating molecule.

Vitamin D3 can help improve hair growth by enhancing the function of specific skin cells and could be useful in hair regeneration treatments.

Some medications might reverse gray hair, especially those that reduce inflammation or stimulate pigment production, and vitamin B might help.

Taking too many vitamin and mineral supplements can cause serious health problems.

Vitamin D and calcium are essential for normal hair growth.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Wound covers with α-13'-COOH from vitamin E can improve and speed up wound healing.