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Vitamin D receptor is important for regulating hair growth and wound healing in mice.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

1,25-(OH)2D3 helps hair grow by blocking certain harmful signals.

MED1 affects skin wound healing differently in young and old mice.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

Recent findings suggest that genetic factors, immune system issues, and skin cell defects might contribute to the development of hidradenitis suppurativa.

Vitamin D might help treat some types of hair loss, but more research is needed.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Vitamin D and calcium are important for quick and effective skin wound healing.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

DNA methylation and long non-coding RNAs are key in controlling hair growth in Cashmere goats.

The mTOR signaling pathway is crucial for hair health and targeting it may lead to new hair loss treatments.

Hair follicles are complex, dynamic mini-organs that help us understand cell growth, death, migration, and differentiation, as well as tissue regeneration and tumor biology.

The conclusion is that certain cell interactions and signals are crucial for hair growth and regeneration.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Some vitamins and minerals like vitamin D and iron can help with certain types of hair loss, but more research is needed for others.

Oxidative stress contributes to hair graying and loss as we age.

Understanding how cells die in the skin is important for treating skin diseases and preventing hair loss.

Certain signals and genes play a key role in hair growth and regeneration, and understanding these could lead to new treatments for skin regeneration.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Cholesterol balance is important for hair health, and problems with it can lead to hair loss conditions.

Genetic research has advanced our understanding of skin diseases, but complex conditions require an integrative approach for deeper insight.