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Scientists created cell lines from balding patients and found that cells from the front of the scalp are more affected by hormones that cause hair loss than those from the back.

A gene mutation in mice causes permanent hair loss and skin issues.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

The Hairless gene is crucial for healthy skin and hair growth.

Hair follicle stem cells are controlled by their surrounding environment.

Smad2/3-dependent TGF-β signaling increases during wound healing.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

BMP signaling controls hair follicle size and cell growth by affecting cell cycle genes.

HPV genes in mice improve ear tissue healing by speeding up skin growth and repair.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

PAD3 plays a key role in hair and skin protein structure and may be linked to skin diseases.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

Neuregulin3 affects cell development in the skin and mammary glands.

Overexpression of HDGF in melanocytes does not cause cancer.

Lactate production is important for activating hair growth stem cells.

MicroRNA-214 is important for skin and hair growth because it affects the Wnt pathway.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Engineered skin substitutes can grow hair but have limitations like missing sebaceous glands and hair not breaking through the skin naturally.

SCF and c-Kit decrease in AGA hair follicles, possibly affecting hair pigmentation and growth.

Hair follicle bulge cells don't help skin regrow after glucocorticoid damage; interfollicular epidermis cells do.

Putting α-methylspermidine on mouse skin can start hair growth.

Transplanting a mix of specific skin cells can significantly improve the repair of damaged hair follicles.

Fat-related cells are important for initiating hair growth.

Certain cells from hair follicles can create new hair and contribute to hair growth when implanted in mice.

Hair follicle dermal stem cells are key for regenerating parts of the hair follicle and determining hair type.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.