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Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.

Estrogens significantly influence hair growth by interacting with receptors in hair follicles and may help regulate the hair growth cycle.

Male pattern baldness involves genetics, hormones, and needs better treatments.

Estrogen therapy can reduce skin aging but has cancer risks.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Vitamin D is important for skin health and may affect conditions like psoriasis and hair loss, but more research is needed to understand its role fully.

Protoporphyrin IX is useful in cancer treatment but can cause health problems if not properly regulated.

TGF-β signaling is crucial for stem cell maintenance, differentiation, and has implications for cancer treatment.

Male pattern baldness involves hormones and cell signals affecting hair growth.

Estrogens help reduce skin aging, and SERMs might offer similar benefits without the risks of hormone therapy.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

The HR protein's role as a repressor is essential for controlling hair growth.

Androgens slow hair growth by altering Wnt signaling in balding cells.

Nuclear hormone receptors play a significant role in skin wound healing and could lead to better treatment methods.

The document concludes that targeting the androgen receptor may be a promising breast cancer treatment, especially for certain types.

Oestrogens help maintain healthy skin, heal wounds, and may protect against skin aging and cancer.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

New findings explain how genetic changes, body clocks, and certain molecules affect tissue response to stress hormones.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

Targeting glucocorticoid action might help treat type-2 diabetes, but human trials are needed.

Estrogens generally inhibit hair growth and improve skin quality, but their exact effects on hair follicles are complex and not fully understood.

The review suggests thorough evaluation and genetic testing for proper diagnosis and treatment of Chrousos syndrome.

Longer CAG repeat lengths in the androgen receptor gene are linked to higher testosterone levels in women with PCOS.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Beard and scalp hair cells have different gene expressions, which may affect beard growth characteristics.

Finger length ratios are not linked to the number of specific gene repeats affecting testosterone sensitivity.

The mineralocorticoid receptor may play a role in skin and hair health and could be a new target for treating related disorders.