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NcoA4 may have roles beyond helping control gene activity, possibly affecting cell behavior and stability.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

The authors maintain that shorter androgen receptor alleles may lead to milder COVID-19 by positively affecting the immune response, not due to changes in testosterone levels or activity.

Mutations in the RBPJ gene cause Adams-Oliver Syndrome.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Certain genetic variations in the AR and ERβ genes can affect androgen levels in women.

The more lipophilic the progesterone derivative, the better it binds to androgen receptors and has antiandrogenic effects.

KLF4 is important for maintaining stem cells and has potential in cancer treatment and wound healing.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

The conclusion is that genetic changes in the glucocorticoid receptor can lead to conditions affecting stress response, immunity, and metabolism, requiring personalized treatment.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

The conclusion suggests that prostate cancer should be classified by castration status and that new therapies targeting androgen receptor signaling show promise.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

The HOXC13 gene affects different hair proteins in cashmere goats in varied ways and is controlled by a feedback loop and other factors.

Wnt proteins from certain skin cells are crucial for normal hair growth and renewal.

Vitamin D receptor helps control hair growth genes in skin cells.

We need better ways to test and understand SARMs to make safer and more effective treatments.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Blocking glucocorticoid receptors improves glucose metabolism in a PCOS mouse model.

The Hairless gene is crucial for healthy skin and hair growth.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.