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Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Foxn1 gene regulation is crucial for thymus development but not for hair growth.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

The skin and thymus develop similarly to protect and support immunity.

FOXN1 is crucial for thymus development and immune response in Xenopus laevis.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

FOXN1 mutations cause severe immunodeficiency, hair loss, nail issues, and thymus defects.

FoxN1 overexpression in young mice harms immune cell and skin development.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

New treatments for immune disorders caused by FOXN1 deficiency are promising.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Researchers made a mouse model with curly hair and hair loss by editing a gene.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

Key genes linked to hair growth and cancer were identified in hairless mice.

A gene mutation in Lama3 is linked to a common type of hair loss.

FOXN1 gene variants cause low T cells and immune issues from birth.

AIRE-deficient rats developed severe autoimmune disease similar to APECED, useful for testing treatments.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Effective hair loss assessment requires a mix of precise measurement methods.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Intestinal intraepithelial lymphocytes are crucial for gut immunity and maintaining the mucosal barrier.

Unconventional lymphocytes are important for quick immune responses and healing of skin and mucosal barriers.

Skin abnormalities can indicate immunodeficiency due to shared origins with the immune system.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

Transplant success has improved with better immunosuppressive drugs and donor matching.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

Proteasome inhibitors are promising treatments for various cancers, autoimmune diseases, and other conditions.

Treg cell-based therapies might help treat hair loss from alopecia areata, but more research is needed to confirm safety and effectiveness.

Two mutations in KRT74 and EDAR genes cause sheep to have finer wool.