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Thermal spring waters and their microbes could be good for skin health and treating some skin conditions in skincare products.

Some cases of tinea capitis, a fungal scalp infection, can look like scarring hair loss due to the body's immune response and the fungus itself.

Keloid skin disorder involves abnormal fibroblast activation and immune response, linked to a group of genes including FGF11.

Pregnancy can cause skin changes and affect existing skin conditions, with limited treatment options due to the need for fetal safety.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

Gray hair can potentially be reversed, leading to new treatments.

Pantethine may boost the immune system's ability to fight sarcoma.

Hyaluronic acid and polycaprolactone improve skin regeneration, with polycaprolactone having a stronger effect on healing and tissue repair.

COVID-19 vaccination may trigger lupus in some people.

IL-17 inhibitors for psoriasis may cause unexpected hair loss.

Janus kinase inhibitors show promise in treating alopecia areata but need more safety research.

Late pregnancy helps repair brain damage in rats due to the GABAergic system.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

Langerhans cells and melanocytes migrate to the skin and hair follicles during early human development.

Alopecia areata patients have higher levels of IL-6 and TNF-α.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

Plasmacytoid dendritic cells are a key factor in causing hair loss in alopecia areata and could help differentiate it from other hair loss conditions.

The secretome from mesenchymal stem cells is a promising treatment that may repair tissue and avoid side effects of stem cell transplantation.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

The document concludes that doctors should recognize congenital triangular alopecia to avoid unnecessary treatments, as it does not respond to steroids like alopecia areata does.

Regulatory T cells are essential for normal and improved wound healing in mice.

The current understanding of frontal fibrosing alopecia involves immune, genetic, hormonal factors, and possibly environmental triggers, but more research is needed for effective treatments.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

Canine pemphigus foliaceus involves significant immune activity and shares similarities with human pemphigus.

In 2017, pediatric dermatology advanced with new treatments and insights into various skin conditions in children.

Genetically repaired stem cells may treat certain genetic diseases, Th17 cells are key in fighting systemic fungal infections, hair loss in AGA is due to progenitor cell loss, and α-synuclein transfer might contribute to Parkinson's disease progression.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

New treatments for Alopecia Areata, like JAK inhibitors, show promise for hair regrowth and are likely to change future treatment approaches.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.