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Mice lacking fibromodulin have disrupted healing patterns, leading to abnormal skin repair and scarring.

New therapies are being developed that target integrin pathways to treat various diseases.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

Prolactin and TGF-β receptor blockers might help treat hair loss.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

TGF-β signaling is crucial for stem cell maintenance, differentiation, and has implications for cancer treatment.

Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.

TGF-β is crucial for controlling stem cell behavior and changes in its signaling can lead to diseases like cancer.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

IGF-1 injections help mice grow more hair by increasing cell growth and blocking a hair growth inhibitor.

TGF-β2 plays a key role in human hair growth and development.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

Eclipta alba extract helps increase hair growth and decrease hair loss-related protein in mice.

Procyanidin compounds from apples and barley promote hair growth and prevent hair cell death.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

Two microRNAs affect hair follicle development in sheep by targeting specific genes.

Patients with Systemic Sclerosis have much higher levels of GDF-15, which could help predict organ involvement and guide treatment.

TNF-alpha inhibitors can cause various immune-related skin issues.

Targeting TGFβ can improve skin immunity in older people.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Smad2/3-dependent TGF-β signaling increases during wound healing.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

The research suggests that p63 and TGF-β1 may help determine tumor type and malignancy in hair follicle and sebaceous tumors.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Estrogen is important for keeping adult mouse nipple skin healthy by controlling certain cell signals.

TGFβ signaling prevents sebaceous gland cells from producing fats.