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The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

Linalool in personal care products may worsen frontal fibrosing alopecia by damaging hair follicle stem cells and triggering harmful immune responses.

Different types of skin cells and immune cells play a role in healing UV-damaged skin, with chronic UV exposure causing lasting damage to certain skin cells.

Different types of sun exposure can damage skin cells and affect healing, with chronic exposure being more harmful, and certain immune cells help in the repair process.

The medicine Cyclosporin A might cause excessive hair growth by reducing a protein that controls hair growth.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

Anti-IL-12/IL-23 antibody therapy effectively treats psoriasiform skin lesions in IBD patients.

Lichen planopilaris may be an autoimmune disease causing hair loss due to immune system issues in hair follicles.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

MDSC-Exo can treat autoimmune alopecia areata and promote hair regrowth in mice.

Follicular mycosis fungoides can look like a B-cell lymphoma, making diagnosis difficult.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

HIV patients with lower CD4 T cell counts often have more skin problems.

Patients with RASopathies are at risk for autoimmune disorders and should be routinely screened.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

The COVID-19 pandemic led to fewer patient visits, more visits by older adults, an increase in cutaneous T-cell lymphoma cases, and a rise in noncosmetic procedures at a dermatology clinic.

Linalool in personal care products may contribute to hair loss by damaging hair follicle stem cells and triggering harmful immune responses.

Aging skin is affected by inflammation, reduced stem cell function, and slower wound healing.

Patients with RASopathies have a higher risk of autoimmune disorders and should be routinely screened.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Rapamycin increased survival in mice with severe chronic graft-versus-host disease by expanding regulatory T cells.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Misbehaving hair follicle stem cells can cause hair loss and offer new treatment options.