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UV light helped human hair transplants survive in mice without broad immunosuppression.

ATIR101 improves survival in stem cell transplant patients; Australian stem cell treatment decisions are influenced by regulation changes.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

A specific enzyme is essential for proper hair follicle stem cell development and healthy skin.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

Researchers found a specific T cell receptor linked to severe drug reactions like Stevens-Johnson syndrome when patients take carbamazepine.

Frontal Fibrosing Alopecia involves disrupted cholesterol pathways, fibrosis, and increased mast cells.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

B cells can both help and hinder the body's defense against melanoma.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Immune cells are essential for skin regeneration using biomaterial scaffolds.

Assessing CD8+ T cell levels before surgery may improve vitiligo treatment outcomes.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Collagen VII helps skin heal and stay strong, sirolimus may lower skin cancer risk in kidney transplant patients, high-molecular-mass hyaluronan helps naked mole rats resist cancer, dermal γδ T cells aid in hair growth in rodents, and overexpression of IL-33 in mouse skin causes itchiness, offering a model for studying allergic inflammation treatments.

New drug targets for hair loss treatment were identified by studying cell interactions in hair follicles.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Cell therapy shows promise for treating severe psoriasis but needs more research to confirm safety and effectiveness.

Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.

Autoimmune hair loss may be linked to increased Th1 and Th17 cells and decreased Th2 cells.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Cellular and immunotherapies show promise for healing chronic wounds but need more research.