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Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

Fibroblast Growth Factors (FGFs) are important for tissue repair and regeneration, influencing cell behavior and other factors involved in healing, and are crucial in processes like wound healing, bone repair, and hair growth.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

Psoriasis involves immune and genetic factors, and understanding these can improve treatments.

New treatments targeting the JAK signaling pathway, especially JAK inhibitors, show promise for alopecia areata.

The document explains how the immune system reacts to organ transplants and the treatments used to prevent rejection.

The Hedgehog Signaling Pathway is important for skin and hair development and skin cancer treatment, but more research is needed to understand it fully.

Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

TSLP affects atopic dermatitis by increasing sebum and reducing fat through IL-4/IL-13 signaling.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Janus kinase inhibitors show promise in treating alopecia areata but need better topical formulations.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Skin's epithelial stem cells are crucial for repair and maintenance, and understanding them could improve treatments for skin problems.

Changing the environment around stem cells could help tissue repair, but it's hard to be precise and avoid side effects.

Different types of skin cells play specific roles in development, healing, and cancer.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

RANK-RANKL signaling is essential for hair growth and skin health.

Single-cell engineered biotherapeutics show promise for skin treatment but need more research and trials.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

New insights into cell communication in psoriasis suggest innovative drug treatments.

Different types of stem cells and their environments are key to skin repair and maintenance.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.