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Valproic Acid could potentially be used to treat immune-related conditions due to its ability to modify immune cell functions.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

Natural killer cells play a key role in autoimmune skin diseases like pemphigus vulgaris.

Inflammation helps stem cells repair tissue by directing their behavior.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

UV light helped human hair transplants survive in mice without broad immunosuppression.

Immune cells are essential for skin regeneration using biomaterial scaffolds.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

B cells can both help and hinder the body's defense against melanoma.

Collagen VII helps skin heal and stay strong, sirolimus may lower skin cancer risk in kidney transplant patients, high-molecular-mass hyaluronan helps naked mole rats resist cancer, dermal γδ T cells aid in hair growth in rodents, and overexpression of IL-33 in mouse skin causes itchiness, offering a model for studying allergic inflammation treatments.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Treg cell-based therapies might help treat hair loss from alopecia areata, but more research is needed to confirm safety and effectiveness.

A substance called miR-1246 may help treat severe hair loss by reducing certain immune cell activities.

T-cell patterns in skin help distinguish alopecia areata from androgenetic alopecia.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

Autoimmune reactions may cause both alopecia areata and HAM.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.

New treatments targeting T-cell pathways are needed for better alopecia areata management.

Autoimmune hair loss may be linked to increased Th1 and Th17 cells and decreased Th2 cells.

ATIR101 improves survival in stem cell transplant patients; Australian stem cell treatment decisions are influenced by regulation changes.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.