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ATIR101 improves survival in stem cell transplant patients; Australian stem cell treatment decisions are influenced by regulation changes.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

The medicine Cyclosporin A might cause excessive hair growth by reducing a protein that controls hair growth.

Different types of skin cells and immune cells play a role in healing UV-damaged skin, with chronic UV exposure causing lasting damage to certain skin cells.

Different types of sun exposure can damage skin cells and affect healing, with chronic exposure being more harmful, and certain immune cells help in the repair process.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

Anti-IL-12/IL-23 antibody therapy effectively treats psoriasiform skin lesions in IBD patients.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

Astilbin can potentially calm overactive immune responses, like in Type 1 Diabetes, by suppressing certain cell activities and reducing inflammation.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Aging skin is affected by inflammation, reduced stem cell function, and slower wound healing.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Rapamycin increased survival in mice with severe chronic graft-versus-host disease by expanding regulatory T cells.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

Misbehaving hair follicle stem cells can cause hair loss and offer new treatment options.

The document doesn't provide a clear conclusion or detailed findings about the impact of certain cells on cancer treatment.

Blocking EGFR in skin cells doesn't majorly increase inflammation markers.

Testosterone helps human pancreatic cells increase insulin release.

MAIT cells may help fight COVID-19 but also contribute to severe inflammation.