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Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Wnt/β-catenin signaling is important for keeping skin cell attachment structures stable.

Dihydrotestosterone treatment on 2D and 3D-cultured skin cells slows down hair growth by affecting certain genes and could be a potential target for hair loss treatment.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Sdr16c5 and Sdr16c6 genes regulate a key point in lipid production that affects eye and skin gland function.

Understanding hair follicle development can help treat hair loss, skin regeneration, and certain skin cancers.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Lysophospholipids like LPA and S1P are important for hair growth, immune responses, and vascular development, and could be targeted for treating diseases.

A man with kidney tumors and lung cysts was diagnosed with Birt–Hogg–Dubé syndrome and treated successfully, with genetic testing confirming the diagnosis.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

A protein found in safflower seeds can stimulate hair growth and speed up wound healing in mice.

Neurotoxins can affect neurotransmitter release and have potential in treating muscle, pain, and cancer conditions, but more research is needed on how they work.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

The nervous system helps control stem cell behavior and immune responses, affecting tissue repair and maintenance.

The research identified key proteins that affect wool fiber thickness in Angora rabbits.

Hair loss from Alopecia Areata is caused by both genes and environment, with several treatments available but challenges in cost and relapse remain.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Keratin 26 affects cashmere goat hair growth and is influenced by various treatments.

A gene mutation in Lama3 is linked to a common type of hair loss.

The document concludes that hair follicle regeneration involves various factors like stem cells, noncoding dsRNA, lymphatic vessels, growth factors, minoxidil, exosomes, and induced pluripotent stem cells.

A new mutation in the ST14 gene broadens the understanding of ichthyosis-hypotrichosis syndrome.

A specific genetic change in the KRT71 gene causes a hair loss condition in Hereford cattle.

The KRTAP36-2 gene in sheep affects wool yield.

Mitochondrial Complex I reduces inflammation and increases bone breakdown by affecting certain immune cells.

Patched1 helps prevent tumors by controlling cell growth.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.