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The study concludes that as skin matures from infancy to childhood, there are major changes in cell differentiation, stemness, and growth, leading to a stronger skin barrier in older children.

Scientists have improved 3D models of human skin for research and medical uses, but still face challenges in perfectly replicating real skin.

EZH2 levels decrease as fetuses develop and are higher in adult skin, which may affect skin growth and repair.

The May issue discussed publishing agreements and four studies on cholesterol in hair, cancer cell changes, hormones in the uterus, and skin protein effects.

Mouse hair follicles age, causing more white hairs due to fewer pigment stem cells.

Skin heals with scars because only one type of fibroblast is used, not a mix.

Fat cells help recruit healing cells and build skin structure during wound healing.

Bone Morphogenetic Proteins (BMPs) help control skin health, hair growth, and color, and could potentially be used to treat skin and hair disorders.

Mice with human skin protein K8 had more skin problems and cancer.

Skin organoids are a promising new model for studying human skin development and testing treatments.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

Fetal skin heals without scarring due to unique cells and processes not present in adult skin healing.

The research reveals how early embryonic mouse skin develops from simple to complex structures, identifying various cell types and their roles in this process.

The study aims to understand the skin and hair characteristics of mothers and their babies, and how these may affect newborns' skin health and mothers' postpartum hair loss.

Newborn skin cells can change into wound-healing cells more easily than adult ones, which might explain why baby skin heals without scars. Understanding this could help treat chronic wounds and prevent scarring.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Skin organoids from stem cells could better mimic real skin but face challenges.

STAT3 signaling is important for healthy skin and hair follicles, and its disruption can lead to skin conditions like atopic dermatitis.

Blocking β-catenin in skin cells improves hair growth during wound healing.

The study mapped yak skin cells to understand hair growth better.

miR-31 regulates hair growth by controlling gene expression in hair follicles.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

cDermo-1 causes dense skin, feathers, and scales in chickens.

Wounds heal without scarring in early development but later result in scars, and studying Wnt signaling could help control scarring.

Helminth protein helps wounds heal better by reducing scarring and promoting tissue growth.

Fetal wounds heal without scarring because of different biological factors, which could help improve adult wound healing.

Hutchinson-Gilford Progeria Syndrome causes rapid aging from a genetic mutation, with no cure but ongoing research into potential treatments.

Mutant mice help researchers understand hair growth and related genetic factors.

Certain transporters are found in human hair follicles and may affect hair growth and loss.

Stem cells are crucial for wound healing and understanding their role could lead to new treatments, but more research is needed to answer unresolved questions.