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Aging causes sweat glands to shrink and move upward, leading to less elastic skin and more wrinkles.

Hormones affect skin aging, and treatments targeting hormonal balance may improve skin health.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

Noggin is necessary to start the hair growth phase in skin after birth.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

The skin's basement membrane has specialized structures and molecules for different tissue interactions, important for hair growth and attachment.

The skin's basement membrane is specially designed to support different types of connections between skin layers and hair follicles.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

The document concludes that understanding hair and feather regeneration can help develop new regenerative medicine strategies.

Smad-4 and Smad-7 are key in hair follicle development, with other Smads being less important.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

Meis1 is crucial for skin health and tumor development.

Chemotherapy and radiation therapy cause skin and hair damage by altering gene expression and signaling pathways.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.

Different processes create patterns in skin and things like hair and feathers.

Experts say proper treatment and sensitivity are important for women's facial skin issues like acne and unwanted hair.

Genetically modified sheep with more β-catenin grew more wool without changing the wool's length or thickness.

The research reveals how early embryonic mouse skin develops from simple to complex structures, identifying various cell types and their roles in this process.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Genes related to keratin, skin cell differentiation, and immune functions are key in hedgehog skin and spine development.

The study found that certain genes are important for hedgehog skin appendage development and immunity, with spines possibly evolving for protection and infection resistance.

BMPs are crucial for hair growth and their decrease by androgens leads to hair loss.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

BMP2 and BMPR-IA may stop hair growth while Noggin may encourage it in yak skin.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

Skin stem cells interacting with their environment is crucial for maintaining and regenerating skin and hair, and understanding this can help develop new treatments for skin and hair disorders.

Hair follicle stem cells are key for hair and skin regeneration, can be reprogrammed, and have potential therapeutic uses, but also carry a risk of cancer.