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The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Wnt activation shows promise for regenerative medicine but requires selective targeting to minimize risks like cancer.

Using developmental signaling pathways could improve adult wound healing by mimicking scarless embryonic healing.

Wnt signaling is crucial for skin and hair development and its disruption can cause skin tumors.

EdnrB signaling helps melanocyte stem cells regenerate and could be targeted to treat pigmentation issues.

OCIAD2 and DCN genes affect hair growth in goats by having opposite effects on a growth signaling pathway and inhibiting each other.

The Notch signaling pathway is important for hair follicle development and could help create treatments for hair disorders.

The HR protein's role as a repressor is essential for controlling hair growth.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

Certain natural and synthetic compounds may help treat inflammatory skin diseases by targeting a specific signaling pathway.

Wnt signaling may be linked to heart diseases in aging and could be a target for future treatments.

The Hairless gene is crucial for hair cell development, affecting whether skin cells become hair or skin and oil gland cells.

Wnt signaling is important for development and cell regulation but can cause diseases like cancer when not working properly.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

A mutation in the KRT71 gene causes a hair disorder by disrupting hair follicle structure and texture.

The PDGF/PDGFR pathway is a potential drug target with mixed success in treating various diseases, including some cancers and fibrosis.

Four genetic risk spots found for hair loss, with WNT signaling involved and a link to curly hair.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

A protein called FERONIA helps control root hair growth in response to cold and low nitrogen by activating nutrient-sensing pathways in a plant called Arabidopsis.

The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.

Cutaneous Lymphadenoma is a unique skin tumor with specific protein markers and common gene mutations that may cause continuous cell growth.

The research provides a gene-based framework for hair biology, highlighting the Hippo pathway's importance and suggesting links between hair disorders, cancer pathways, and the immune system.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

Editing the FGF5 gene in sheep increases fine wool growth.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Mutations in the NR3C1 gene cause a rare condition that affects hormone signaling and can lead to various symptoms, with dexamethasone as a treatment option.

Stem cells compete for space using cell adhesion, and mutations can affect their competitive success, with implications for tissue health and disease.

EDA signaling is linked to skin disorders, various cancers, and liver disease.