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The conclusion is that a treatment called cp-asiAR can reduce hair loss and promote hair growth, making it a potential new therapy for androgenetic alopecia.

The QuantAnts machines can find cancer markers and create CRISPR targets for them.

Targeted siRNA therapy may be a promising treatment for KID syndrome by reducing mutant gene expression and improving cell communication.

cp-asiAR may effectively treat hair loss by targeting androgen receptors.

cp-asiAR may effectively treat androgenetic alopecia by promoting hair growth and reducing androgen receptor activity.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Different types of skin cells are organized in a special way in large wounds to help with healing and hair growth.

The hair keratin gene KRT81 is found in both normal and breast cancer cells and helps them invade surrounding tissues.

TGF-β1 increases androgen receptor activity in hair loss, but Hic-5/ARA55 can counter this effect.

Vitamin D boosts a specific protein in skin cells linked to hair follicles.

Gene regulation could revolutionize hair color by altering pigmentation from within.

The study found new details about human hair growth and suggests that preventing a specific biological pathway could potentially treat hair graying.

EDA and EDAR are important for hair follicle development in cashmere goats and affect other related genes.

Megestrol acetate helps fat-derived stem cells grow, move, and turn into fat cells through a specific receptor.

The research identified genes that explain why some sheep have curly wool and others have straight wool.

HSP90 and lamin A/C are crucial for hair growth and could be targets for treating hair loss.

Blocking certain immune signals can reduce skin damage from radiation therapy.

A circular RNA helps cashmere goat hair cells become hair follicles by blocking a molecule to boost a gene important for hair growth.

The OVOL1 gene, controlled by β-catenin, is crucial for creating hair follicles.

Self-amplifying RNA could be a better option for protein replacement therapy with lower doses and lasting effects, but delivering it into cells is still challenging.

Gene therapy shows promise for improving wound healing, but more research is needed for human use.

Notch1 signaling is crucial for improving wound healing and skin regeneration by affecting stem cell behavior.

Reducing FOXA2 in skin cells lowers their ability to grow hair.

Scientists can mimic hair disorders by altering genes in lab-grown human hair follicles, but these follicles lack some features of natural ones.

Certain genes controlled by OVOL1 are crucial for creating new hair follicles.

Higher levels of the DP2 receptor may lead to hair loss.

The gene HDC is important for the development of hair follicles in newborn mice.

Annexin A2 isoform 2 helps dermal papillae cells grow, affecting hair growth.

CCL5 is important for the hair growth potential of human dermal papilla cells.