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Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.

A protein called sFRP4 can slow down hair regrowth.

sFRP4 partially inhibits hair regeneration, but the study needs clearer data analysis and better explanation of the process.

A protein called sFRP4 can partly inhibit hair growth.

Poor maternal nutrition can lead to fewer wool follicles in Chinese Merino sheep.

SFRP2 and PTGDS may be key factors in female hair loss.

The research suggests that SFRP2 and PTGDS proteins might be indicators of female pattern hair loss and could contribute to hair loss.

Finasteride exposure affects gene expression and anogenital distance in male rat fetuses.

Hair loss can be caused by stress, infections, drugs, and various diseases, with treatment depending on accurate diagnosis.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Two main types of fibroblasts with unique functions and additional subtypes were identified in human skin.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

Hair stem cell regeneration is controlled by signals that can explain different hair growth patterns and baldness.

Blocking JAK-STAT signaling can lead to hair growth.

The WNT signaling pathway is important in many diseases and targeting it could offer new treatments.

Exosomes could potentially enhance tissue repair and regeneration with lower rejection risk and easier production than live cell therapies.

Blocking the Wnt pathway could lead to new treatments for cancer and tissue repair but requires careful development to avoid side effects.

Olive oil compounds may help prevent cancer in animals, but human results are mixed.

Stress hormone corticosterone blocks a growth factor to slow down hair stem cell activity and hair growth.

The conclusion is that Fgf18 and Tgf-ß signaling could be targeted for hair loss treatments.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

The document concludes that for hair and feather growth, it's better to target the environment around stem cells than the cells themselves.

Skin fat has important roles in hair growth, skin repair, immune defense, and aging, and could be targeted for skin and hair treatments.

The document concludes that understanding hair and feather regeneration can help develop new regenerative medicine strategies.

Environmental factors at different levels control hair stem cell activity, which could lead to new hair growth and alopecia treatments.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Skin tumor regression is helped by retinoic acid signaling blocking Wnt signaling.

Hair follicles can help wounds heal faster and this knowledge could be used to treat chronic skin ulcers, with a potential use of a special stem cell hydrogel to enhance healing.