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Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

Stress stops hair growth in mice by causing early hair growth phase end and harmful inflammation through a specific nerve-related pathway.

Minoxidil helps prevent stress-caused hair loss in mice.

Squalene may be a marker for certain types of alopecia.

Stress may affect hair growth by influencing hair follicle development and could contribute to hair loss.

Immune cells around hair follicles help control hair growth and could be targets for treating hair disorders.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

Hair loss in mice starts with immune cells damaging hair roots before it becomes visible.

Quercetin significantly helps hair growth by activating hair follicles and improving blood vessel formation around them.

Cholesterol-related compounds can stop hair growth and cause inflammation in a type of scarring hair loss.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Telocytes might help with skin repair and regeneration.

Stress increases a factor in mice that leads to hair loss, and blocking this factor may prevent it.

Macrophages are vital for skin healing, hair growth, salt balance, and cancer defense.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

FOL-026 peptide can help repair blood vessels and promote growth, offering potential treatment for vascular diseases.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

The document concludes that for hair and feather growth, it's better to target the environment around stem cells than the cells themselves.

Understanding how hair follicle stem cells work can help find new ways to prevent hair loss and promote hair growth.

Immune cells help regulate hair growth, and better understanding this can improve hair loss treatments.

Immune cells affect hair growth and could lead to new hair loss treatments.

Sebaceous glands play a key role in skin health, immunity, and various skin diseases.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Hair follicles are essential for skin health, aiding in hair growth, wound healing, and immune function.

Increased PPARGC1α relates to hair thinning in common baldness.