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Two rare bald spots on the back of the scalp were found to be lupus, not alopecia areata.

The document concludes that ongoing research using animal models is crucial for better understanding and treating Alopecia Areata.

The article concludes that schoolchildren and adolescents experienced various skin issues during the COVID-19 pandemic, including acne from masks and other skin reactions from the virus and vaccines.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Alopecia Areata is an autoimmune condition causing hair loss with no cure and treatments that often don't work well.

Lupus is a complex disease that requires personalized treatment because it varies greatly between individuals.

Certain genes on chromosomes 6, 10, 16, and 18 may increase the risk of alopecia areata.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Finasteride and minoxidil are effective for hair regrowth, while treatments for alopecia areata have varying success and continuous treatment is necessary.

Thyroid disease can cause hair loss and treating thyroid problems might help with hair disorders.

A woman with systemic sclerosis developed a unique scarring hair loss combining features of systemic sclerosis and frontal fibrosing alopecia.

Syphilitic alopecia is a rare hair loss condition in secondary syphilis that looks similar to another condition but can be diagnosed with specific tests and responds to antibiotics.

The document describes how to tell different types of non-scarring hair loss apart by looking at hair and scalp tissue under a microscope.

Accurate diagnosis of pseudolymphomatous folliculitis is crucial to avoid mistaking it for more serious conditions.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

The research confirms that Hidradenitis Suppurativa is characterized by increased inflammation, disrupted skin cell organization, and abnormal metabolic processes.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Transplanted hair follicles can change and adapt to new areas of the body, with the immune system possibly playing a role in this adjustment.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Stress can cause hair loss by affecting nerve-related hair growth, and noradrenaline might help prevent this.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

The C-allele and CC-genotype in the PTPN22 gene lower the risk of alopecia areata.

Skin can produce blood cells, often due to disease, which might lead to new treatments for skin and blood conditions.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

NK cells play a role in skin diseases like eczema and psoriasis.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Mogamulizumab, a skin cancer drug, may cause hair loss similar to alopecia areata.

Targeting Vδ1+T-cells may help treat alopecia areata.