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Finasteride improves heart function in obese and aging male rats by reducing oxidative stress and p53 protein levels.

A chemical called 5-Bromo-2′-deoxyuridine caused rapid hair loss in mice by killing certain skin cells through a specific cell death pathway.

The Hr protein binds to DNA, interacts with p53, and affects cell cycle genes.

TRH stimulates human hair growth and extends the hair growth phase.

A specific ATR gene mutation is linked to a hereditary oropharyngeal cancer syndrome.

Inactive hair follicle stem cells help prevent skin cancer.

Keratin 17 is linked to various diseases, including cancer and skin conditions, and may be a target for diagnosis and treatment.

Apoptosis is crucial for healthy skin and treating skin diseases.

Cyclosporin A helps mice grow hair by blocking a specific protein activity in skin cells.

WNT10A helps esophageal cancer cells spread and keep renewing themselves.

The S100A4 protein is more common in psoriatic skin and could be a target for treating psoriasis.

Ginsenoside Rb1 slows down aging in mice by affecting cell growth, cell death, and metabolism.

Short-term finasteride treatment may lower some cell death factors in prostate cancer cells.

HPV genes in mice improve ear tissue healing by speeding up skin growth and repair.

The photolyase-based device significantly changed the size and heat of potential skin cancer areas in patients.

Certain blood miRNAs are linked to severe alopecia areata and could lead to new treatments.

The study found new details about human hair growth and suggests that preventing a specific biological pathway could potentially treat hair graying.

Formononetin, found in red clover plants, can promote hair regrowth and reduce hair loss by preventing cell death.

Tetrahydroxystilbene Glucoside may help prevent hair loss by blocking certain pathways that lead to cell death.

Chemotherapy may cause recurring hair loss due to an autoimmune response.

Ginsenoside Rg1 protects mouse skin from UVB damage and helps control inflammation.

Keratin 17 is important for skin's response to radiation, affecting many genes and cell division.

The conclusion suggests that focusing on certain cellular pathways may improve the prevention and repair of hair loss caused by radiotherapy.

Genes related to keratin, skin cell differentiation, and immune functions are key in hedgehog skin and spine development.

Grasp protein helps maintain skin health after UVB exposure.

The document concludes that understanding how hair follicles naturally die and regenerate is important for insights into organ development and could impact health and disease treatment.

The debate focuses on the role of catagen and hair shedding mechanisms in telogen effluvium.

Mild oxidative stress can prevent hair loss from chemotherapy.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.