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Research 31–60 of 495
- 331 Mitochondrial stress contributes to atopic dermatitis
- Decision letter: Involvement of ILC1-like innate lymphocytes in human autoimmunity, lessons from alopecia areata
- 098 Simvastatin decreases AA-associated inflammation through effects on isoprenoid metabolites
- 080 Mouse models of alopecia areata: C3H/HeJ mice versus the humanized AA mouse model
- 원형탈모증(alopecia areata)의 최신 이해와 치료
- 328 The transcription factor CEBPB is a novel hub gene and multi-functional disease driver in Psoriatic skin inflammation
- LB708 ILC1-like innate lymphocytes in human autoimmunity: Lessons from Alopecia Areata
- The immunopathogenesis of Alopecia Areata
- CD4+ Treg expansion arrests disease progression in a pre-clinical model of alopecia areata
- Alopecia areata: a review of disease pathogenesis
- Treatment of alopecia areata: “What is new on the horizon?”
- 516 Possible role of ILC1 in the pathogenesis of alopecia areata (AA)
- Getting to the root of hair loss in alopecia
- Alopecia areata: from immunopathogenesis to emerging therapeutic approaches
- <i>IKZF1</i> and Ikaros Overexpression Results in Alopecia Areata‐Like Phenotype in Mice
- 056 IKZF1 and IKAROS overexpression contributes to the pathogenesis of alopecia areata
- Genes and Immune Response in Alopecia Areata: Review of the Alopecia Areata Research Summit First Day Proceedings
- Pathophysiology of Alopecia Areata in the Pediatric Patient
- Prior antigen experience influences the development of alopecia areata in C3H/HeJ mice 2258436
- Pathomechanisms of immune-mediated alopecia
- 649 Gut microbiota plays a role in the development of alopecia areata
- Alopecia Areata
- Mathematical model for alopecia areata
- Janus Kinase Inhibitors against Other Biological Treatments in Alopecia Areata
- ULBP3: a marker for alopecia areata incognita
- Inhibiting Janus kinases to treat alopecia areata
- Innate lymphoid cells type 1 may be new, non-antigen-specific player in the pathogenesis of alopecia areata
- Alopecia areata is characterized by dysregulation in systemic type 17 and type 2 cytokines, which may contribute to disease‐associated psychological morbidity
- Involvement of ILC1-like innate lymphocytes in human autoimmunity, lessons from alopecia areata
- Butyrophilin-like 2 (BTNL2) to the Rescue: controlling autoaggressive dermal gamma delta (GD) T lymphocytes in human hair follicles (HFs) 3747