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Urokinase, a type of protein, helps skin cells multiply faster, especially in newborn mice.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

The document concludes that understanding how the skin's immune system and inflammation work is complex and requires more research to improve treatments for skin diseases.

Stem cells rearrangement regenerates functional hair follicles, potentially treating hair loss.

Skin organoids are a promising new model for studying human skin development and testing treatments.

Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

Vitamin D helps protect skin, PSORS1 gene's risk interval for psoriasis is expanded, hair follicles can be generated from mouse cells, and interferon-γ may cause pigmented skin lesions.

Engineered skin substitutes can grow hair but have limitations like missing sebaceous glands and hair not breaking through the skin naturally.

Hair follicles have a more inactive cell cycle than other skin cells, which may help develop targeted therapies for skin diseases and cancer.

Estrogen is important for keeping adult mouse nipple skin healthy by controlling certain cell signals.

Patient-derived melanocytes can potentially treat vitiligo by restoring skin pigmentation.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

BMP signaling is essential for the development of touch domes.

Activating a protein called β-catenin in adult skin can make it behave like young skin, potentially helping with skin aging and hair loss.

Using skin stem cells and certain molecules might lead to scar-free skin healing.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

Nonionic liposomes are the best for delivering genes to skin cells.

Understanding how melanocyte stem cells work could lead to new treatments for hair graying and skin pigmentation disorders.

Different species and human skin models vary in their skin enzyme activities, with pig skin and some models closely matching human skin, useful for safety assessments and understanding the skin's protective roles.

Hair follicle bulge cells don't help skin regrow after glucocorticoid damage; interfollicular epidermis cells do.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Fat stem cells from diabetic mice can help heal skin wounds in other diabetic mice.

Three specific proteins can turn adult skin cells into hair-growing cells, suggesting a new hair loss treatment.

Hair follicle regeneration in skin grafts may be possible using stem cells and tissue engineering.

The document concludes that there is a significant lack of reporting on the sex and age of cells in skin research, which could affect clinical trials and treatments.

Inhibiting class I HDACs helps maintain hair growth ability in skin cells.

New methods for skin and nerve regeneration can improve healing and feeling after burns.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.