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Minoxidil and platelet-rich plasma can help turn thin hair into thicker hair in male pattern baldness.

New method efficiently isolates hair growth cells from newborn mouse skin.

The study found that stem cells and neutrophils are important for regenerating hair follicle structures in mice.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

Two mouse mutants have defective hair cuticle cross-linking.

Vitamin D3 applied to mouse skin caused more wrinkles and sagging due to changes in the skin's outer layer.

Copper treatments increase copper in all tissues, but brindled female mice accumulate much more copper in their kidneys without clinical effects, unlike brindled male mice where brain copper deficiency is clinically significant.

PRP helps hair regrowth and thickness.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Diet changes can protect against harmful environmental effects on fetal development.

Minoxidil can help grow hair in mice by making cells grow and improving hair quality. More research needed.

Understanding the mouse hair cycle is crucial for cancer research.

Lanceolate complexes in mouse hair follicles are essential for touch and depend on specific cells for maintenance and regeneration.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Mice healed without scars as fetuses but developed scars as adults, suggesting scarless healing might be replicated with further research.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Nude mice with grafted human skin developed scars similar to human hypertrophic scars.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Bezafibrate treatment improved skin and spleen health in aging mice but didn't extend lifespan.

S100A3 protein is crucial for hair shaft formation in mice.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

New hair growth corticosterone levels are higher in diabetic mice, indicating long-term stress.