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Hair follicle bulge cells are important for hair survival and help heal the skin after injury, which might be relevant for understanding hidradenitis suppurativa.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

Mutations in the HEPHL1 gene cause abnormal hair and cognitive issues.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Quercetin effectively treated and prevented hair loss in mice.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

Neutrophils are key in causing chronic itch in atopic dermatitis, and blocking CXCR3 could reduce this itch.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

The project created a standardized system for classifying skin lesions in lab rats and mice.

Cidea is essential for proper lipid storage and secretion in sebaceous glands, affecting skin and hair health.

Baby teeth stem cells can help grow hair in mice.

miR-29 is a key factor that accelerates aging.

Hydrogel scaffolds can help wounds heal better and grow hair.

sPLA2-X is crucial for normal hair growth and follicle health.

Hair follicle stem cells reduced hair loss and inflammation in mice with a condition similar to human alopecia.

A protein called desmoglein 3 is important for keeping hair follicle stem cells inactive and helps in their regeneration.

Disrupting a specific protein's function in hair follicle stem cells triggers their activation and a self-healing process.

Aging reduces skin cell renewal and defense against germs due to TGFbeta, but blocking TGFbeta could help restore these functions.

Hair grows faster in the morning and is more vulnerable to damage from radiation due to the internal clock in hair follicle cells.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

The study found that diseases can be grouped by symptoms and that the accuracy of predicting disease-related genes varies with the data source.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

LEF1 is essential for the development of airway glands and is regulated by the Wnt/ß-catenin pathway.

Hair follicle regeneration is possible but challenging, especially in humans, due to the need for specific cells and a better understanding of how they signal growth.

Hair follicle cells help maintain and support stem cells and blood cell formation.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

Human skin xenografting could improve our understanding of skin development, renewal, and healing.

Removing UBE2N from skin cells causes inflammation and immune response, which can be lessened with specific inhibitors.

Mouse skin glands need healthy nerves to grow properly during hair growth phases.