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Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

Progeroid mouse models show signs of early aging similar to humans, helping us understand aging better.

Alopecia areata can be triggered by specific immune cells without genetic or environmental factors.

Both mouse and rat models are effective for testing alopecia areata treatments.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Mice with human gene experienced hair loss when treated with DHT.

The HCR gene contributes to psoriasis risk.

Ion-paired risedronate significantly increases skin penetration without irritation compared to risedronate alone.

A new mouse model helps understand and find treatments for alopecia areata.

A mouse gene mutation increases the risk of skin cancer.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

A new model for hair regeneration in mice was created in 2015, which is faster and less invasive than the old method, producing normal hairs in about 21 days.

Akt2 protein is essential for normal cell division in early mouse embryos.

Cryopreserved mouse whisker follicles can grow hair when transplanted into nude mice.

Two mouse mutations cause similar hair loss despite different skin changes.

Vesicles from irradiated mouse cheek skin help cells survive radiation.

Minoxidil absorption in skin is slowed by cleansing, depends on how long it stays on the skin, and is not much affected by reapplication.

Scientists created complete hair-like structures by growing mouse skin cells together in a special gel.

The mouse model suggests male pattern baldness may be due to an enzyme increasing DHT and higher androgen receptor levels in hair follicles.

The mouse models are effective for testing new hair loss treatments.

Minoxidil and platelet-rich plasma can help turn thin hair into thicker hair in male pattern baldness.

New method efficiently isolates hair growth cells from newborn mouse skin.

The study found that stem cells and neutrophils are important for regenerating hair follicle structures in mice.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

Two mouse mutants have defective hair cuticle cross-linking.

Vitamin D3 applied to mouse skin caused more wrinkles and sagging due to changes in the skin's outer layer.

Copper treatments increase copper in all tissues, but brindled female mice accumulate much more copper in their kidneys without clinical effects, unlike brindled male mice where brain copper deficiency is clinically significant.