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Stress might contribute to hair loss in alopecia areata.

Alopecia areata is an incurable autoimmune condition causing hair loss, with research aiming for better treatments.

People on immune-modifying skin disease treatments may have a weaker antibody response to COVID-19 vaccines but often improve after the second dose.

Somatostatin may help protect hair follicles from immune attacks.

Certain gene variations in EGF and EGFR may increase the risk of alopecia areata in Koreans.

Oral tofacitinib can treat both psoriasis and alopecia universalis by normalizing inflammatory pathways.

Higher antigliadin antibodies are linked to more severe alopecia areata, suggesting screening for celiac disease in these patients.

Higher antigliadin antibody levels are linked to alopecia areata severity.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

Early treatment with immunoglobulin and aspirin reduces heart complications in children with Kawasaki disease.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Half of people with Alopecia Areata may see hair regrowth within a year without treatment, but recovery is unpredictable.

The 2003 guidelines suggest that while some treatments can regrow hair in alopecia areata, none alter the disease's progression, and wigs may be the best option for extensive hair loss.

Alopecia areata is an autoimmune disease causing patchy hair loss, often with other autoimmune disorders, but its exact causes are unknown.

Folliculitis decalvans is a rare scalp condition causing scarring hair loss, treated with long-term antibiotics and other medications, but it often comes back and is hard to manage.

Alopecia areata can cause unpredictable hair loss, and treatments like corticosteroids and minoxidil may help but have varying side effects.

Women with PCOS have fewer activated T cells in their ovarian follicles, which might affect fertility.

Despite progress in treatment, the exact cause of Alopecia areata is still unknown.

Plasmacytoid dendritic cells are a key factor in causing hair loss in alopecia areata and could help differentiate it from other hair loss conditions.

Modified rat stem cells on a special scaffold improved blood vessel formation and wound healing in skin substitutes.

Tofacitinib citrate improved nail dystrophy and pain in patients with alopecia universalis without causing side effects.

Interferons are effective for some skin conditions and cancers, but can have side effects and need more research for optimal use.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Interferon gamma alone can't cause alopecia areata in C3H/HeJ mice.

Targeting immune tolerance issues in Alopecia Areata could restore hair growth and maintain remission.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Fractional lasers and microneedling, combined with topical agents, could potentially treat Alopecia Areata effectively, but more research is needed due to limited data.

A genetic marker linked to a type of hair loss was found in most patients studied.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.