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Hair and skin can regenerate without bulge stem cells due to other compensating cells.

Animal models, especially mice, are essential for advancing hair loss research and treatment.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

A pathway helps maintain long telomeres in both stem and cancer cells.

The timing of when the gene Bmal1 is active affects aging and survival, with its absence during development, not adulthood, leading to premature aging.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Hair follicles produce and respond to melatonin, affecting hair growth and sensitivity to estrogen.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

μ-Crystallin may help hair growth by affecting thyroid hormone levels in mouse hair follicles.

Activin A and follistatin control when hair cells develop in mouse ears.

Gene therapy in mice increased lifespan and improved health without causing cancer.

Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

Mice with enhanced regeneration abilities may help develop new regenerative medicine therapies.

Changing the diet of mice lacking the enzyme CBS can affect symptoms related to the genetic condition.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

MPA increases cancer spread by boosting Eph A2 activity.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

The skin's basement membrane has specialized structures and molecules for different tissue interactions, important for hair growth and attachment.

15-lipoxygenase helps keep skin healthy by reducing inflammation.

New treatments for diabetes, central nervous system repair, and cartilage injury were found, and a way to create functional hair follicles from stem cells was developed.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Researchers successfully transplanted hair follicles in mice, which survived well and helped in wound healing.

The skin's basement membrane is specially designed to support different types of connections between skin layers and hair follicles.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.