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Topical treatments can deliver active molecules to skin stem cells, potentially helping treat skin and hair disorders, including skin cancers and hair loss.

Acne is a common skin condition linked to diet, hormones, and genetics, and early treatment can prevent scarring.

PCOS has a strong genetic basis, but more research is needed to fully understand it.

Stingless bee propolis at a 6.25% concentration effectively stops dandruff-causing bacteria growth, performing better than ketoconazole.

Human hair keratins can self-assemble and support cell growth, useful for biomedical applications.

The study found that sweat glands normally suppress immune responses, but this is disrupted in certain skin diseases, possibly contributing to their development.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Sebocytes play a key role in controlling androgen levels in human skin.

Modulating Cytochrome P450 activity could help develop new skin disease treatments.

Prolactin affects hair growth and skin conditions, and could be a target for new skin disease treatments.

Hairless protein reduces Msx2 gene activity, affecting hair follicle development.

Sodium metabisulfite increases sodium channel activity, leading to higher cell excitability and potential damage.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Minoxidil sulfotransferase is a marker of keratinocyte differentiation and may play a role in hair growth.

Canine pemphigus foliaceus involves significant immune activity and shares similarities with human pemphigus.

Geranium Sibiricum L extract may promote hair growth by increasing growth factors and decreasing inhibitory factors under stress.

Androgenetic alopecia is mainly caused by genetic factors and increased androgen activity, leading to hair follicle miniaturization.

Eyelid cells share signaling components but differ in pathway activity.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Activating the Nrf2 pathway reduces inflammation and cell activation in human hair follicles, suggesting a potential treatment for certain hair loss conditions.

The analysis of a large pilomatricoma revealed five distinct areas with different gene activity related to hair growth and tumor development.

Terbinafine and itraconazole are more effective against Microsporum canis than fluconazole and griseofulvin.

Different lab conditions and light treatment methods change how human skin cells respond to light therapy.

Antiandrogens are effective for conditions like prostate cancer and skin issues, but more research is needed to confirm their benefits and minimize side effects.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

Different human hair follicle stem cells grow at different rates and respond differently to a baldness-related compound.

New pregnane derivatives were more effective than finasteride at inhibiting a key enzyme for male pattern baldness.

Oxidative stress affects hair loss in men with androgenetic alopecia.