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Finasteride effectively inhibits 5α reductase, while plant extracts like Permixon and Bazoton don't show significant results.

Finasteride exposure in pregnancy causes genital abnormalities in male rats.

Men over 66 taking medication for prostate enlargement have a higher risk of depression and self-harm, especially in the first 18 months of treatment.

Cancer patients treated with immune checkpoint inhibitors may develop alopecia, but some hair regrowth is possible with treatment.

Finasteride affects male rat genitalia development, causing abnormalities during specific pregnancy days.

5α-reductase inhibitors can cause sexual problems, higher risk of aggressive prostate cancer, and depression.

Finasteride effectively reduces hair growth in women with idiopathic hirsutism, but requires careful contraception during treatment.

5-alpha reductase inhibitors can cause sexual side effects like erectile dysfunction and reduced sexual desire, sometimes lasting after stopping the drug.

Finasteride reduces hairiness and androgen levels in women with unexplained excessive hair growth.

The conclusion is that Fgf18 and Tgf-ß signaling could be targeted for hair loss treatments.

Finasteride, when given in high oral doses to pregnant monkeys, caused genital abnormalities in male fetuses, but not in female fetuses or those exposed to intravenous finasteride.

Baricitinib is effective and safe for treating severe alopecia areata.

Men taking 5α-reductase inhibitors for enlarged prostate have a higher chance of experiencing reduced sexual desire and erectile dysfunction.

Finasteride and dutasteride effectively treat hair loss, but may cause side effects like sexual dysfunction and depression.

Finasteride almost fully depletes allopregnanolone in rat brains and enhances 20α-DHP, but doesn't change 3α-DHP levels.

Both drugs lower DHT levels, with GI198745 being more effective.

Finasteride reduces scalp DHT levels, potentially treating male pattern baldness.

5α-reductase inhibitors increase the risk of sexual dysfunction, especially in men with enlarged prostate.

Finasteride reduces hair loss and treats BPH without major hormone changes, but may cause sexual dysfunction.

Antidepressants called SSRIs can cause skin problems, bleeding risk, and other side effects.

Antidepressants, 5α-reductase inhibitors, and isotretinoin can cause long-lasting sexual dysfunction.

Blocking a specific enzyme in male rat fetuses leads to the development of nipples and feminized genitalia.

Using finasteride or dutasteride may cause long-lasting erectile dysfunction.

Exemestane may protect bones by stimulating osteoblast growth through androgen-related pathways.

Finasteride, a drug, can block the seizure-preventing effects of a hormone called progesterone in mice.

Long-term finasteride use doesn't change bone density or metabolism.

A new drug, dutasteride, is at least as effective as the older drug, finasteride, for treating enlarged prostate and may have additional uses.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

Four new compounds were more effective than finasteride in treating prostate issues and hair loss, with one being 100 times more active and safe for use.

5-α-reductase inhibitors reduce prostate cancer risk but may cause sexual dysfunction and don't affect high-grade tumor or death rates.