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New insights into cell communication in psoriasis suggest innovative drug treatments.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

Human hair keratins can self-assemble and support cell growth, useful for biomedical applications.

Scientists successfully made a human growth factor in a plant, which could help with hair growth and bone development.

Finasteride in male rats causes liver and metabolic issues in their offspring.

Notch signaling disruptions can cause various skin diseases.

Myotonic Dystrophy may age cells faster, and drugs that target aging could be potential treatments.

Recognizing specific skin features helps diagnose and manage lupus erythematosus effectively.

Long-haul COVID can cause lasting symptoms affecting many body systems and may be linked to ongoing inflammation and immune system issues.

Humans and pets share similar allergy mechanisms, and studying pet allergies can help treat both human and animal allergies.

Catalytic antibodies are early indicators and active participants in the development of systemic lupus erythematosus.

The study found key factors in the cause of hidradenitis suppurativa, its link to other diseases, and identified existing drugs that could potentially treat it.

Menopause causes significant changes in the vaginal introitus, but less so in the labia majora, which may lead to symptoms of vaginal atrophy.

The 2015 Hair Research Congress concluded that stem cells, maraviroc, and simvastatin could potentially treat Alopecia Areata, topical minoxidil, finasteride, and steroids could treat Frontal Fibrosing Alopecia, and PTGDR2 antagonists could also treat alopecia. They also found that low-level light therapy could help with hair loss, a robotic device could assist in hair extraction, and nutrition could aid hair growth. They suggested that Alopecia Areata is an inflammatory disorder, not a single disease, indicating a need for personalized treatments.

The guide explains how to study human and mouse sebaceous glands using various staining and imaging techniques, and emphasizes the need for standardized assessment methods.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

Genes linked to fibrosis are more active in people with central centrifugal cicatricial alopecia.

Skin's epithelial stem cells are crucial for repair and maintenance, and understanding them could improve treatments for skin problems.

Epidermal stem cells show promise for future dermatology treatments due to ongoing advancements.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.

Fibroblast Growth Factors (FGFs) are important for tissue repair and regeneration, influencing cell behavior and other factors involved in healing, and are crucial in processes like wound healing, bone repair, and hair growth.

Hair follicle stem cells are a promising source for tissue repair and treating skin or hair diseases.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Skin healing from blisters can delay hair growth as stem cells focus on repairing skin over developing hair.

Sebaceous glands can help harvest hair follicle stem cells to regenerate skin and hair.

When skin blisters, healing the wound is more important than growing hair, and certain stem cells mainly fix the blisters without helping hair growth.

Acne is linked to inflammation and insulin resistance, and is associated with various syndromes that require different treatments.

The document concludes that certain genetic mutations and dietary factors are involved in acne development, and treatments like isotretinoin and diet changes can help manage it.

Male genital development is driven by androgen signaling and understanding it could help address congenital anomalies.

A woman's hair turned white after taking a cancer drug called dasatinib.