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The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

TAF4 is important for skin cell growth and helps prevent skin cancer in mice.

New materials that better mimic natural skin structure could improve healing, especially for chronic wounds.

α-parvin is necessary for skin and hair growth and for the correct orientation of skin cells.

Deleting MPZL3 increases skin oil production and reduces body fat.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

miR-31 regulates hair growth by controlling gene expression in hair follicles.

Epidermal stem cells are diverse and vary in activity, playing key roles in skin maintenance and repair.

UVB exposure increases skin proteins for retinoic acid synthesis and shifts their location, possibly affecting skin repair.

Cathepsin L is essential for normal hair growth and development.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Injecting follicular cells into skin can lead to the formation of new hair follicles.

Inhibiting ornithine decarboxylase may help prevent certain skin cancers.

Targeting ornithine decarboxylase can help prevent skin cancer.

High ODC and low K1 and K10 may indicate early skin tumors in mice.

A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

KLF4 is important for keeping hair follicle stem cells inactive.

Blocked hair follicles cause acne, and studying tiny early acne spots helps understand its severity.

Patched1 helps prevent tumors by controlling cell growth.

A topical BRAF inhibitor, vemurafenib, can speed up wound healing and promote hair growth, especially in diabetic patients.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

FA2H is essential for normal fur and sebum production in mice.

The type 3 IP3 receptor is important for controlling hair loss and growth.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

The gene Foxq1, controlled by Hoxc13, is crucial for hair follicle differentiation.

Hair can regrow after certain stem cells are lost because other stem cells can take over their role.

Different types of long-lasting stem cells are responsible for the growth and upkeep of the mammary gland.

Lactate production is important for activating hair growth stem cells.

Jumonji genes are important for development and their mutations can cause abnormalities, especially in the heart and brain.