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Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Bald thigh syndrome in sighthounds is caused by structural defects in hair shafts due to downregulated genes and proteins.

Certain transcription factors are key in controlling skin stem cell behavior and could impact future treatments for skin repair and hair loss.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

The document reports findings on genetic research, including ethical concerns about genome editing, improved diagnosis of mitochondrial mutations, solving inherited eye diseases, confirming gene roles in epilepsy, linking a gene to aneurysms, and identifying genes associated with age-related macular degeneration.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

Histone demethylases play a key role in the development of many diseases and may be targets for treatment.

JAGGED1 could help regenerate tissues for bone loss and heart damage if delivered correctly.

A new genetic change in the DSC3 gene is linked to a rare condition causing hair loss and skin blisters in a child.

Researchers found a genetic link for hereditary hair loss but need more analysis to identify the exact gene.

The genes KRT25 and SP6 affect curly hair in horses, with KRT25 also causing hair loss. If both genes are mutated, the horse gets curly hair and hair loss. KRT25 can hide the effect of SP6.

Hairless protein affects hair follicle structure by regulating the Dlx3 gene.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

The transcription factor Meis2 is essential for touch sensation and proper nerve development in touch receptors.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Dlx3 is essential for hair growth and regeneration.

Chromosomal changes, including those in the WRN gene and rDNA, may significantly contribute to aging.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

Mutations in NIPAL4 cause skin issues by disrupting lipid layers, but some improvement is seen with topical treatment.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

Hoxc13 gene affects wool length in Gansu alpine fine-wool sheep.

Genes related to calcium signaling and lipid metabolism are important for curly hair in Mangalitza pigs.

Cutaneous gene therapy could become a viable treatment for skin and hair disorders with improved vector development and gene expression control.

SOX9 is essential for the development of various organs and hair follicles.

Sox2 controls hair color by affecting pigment production in hair follicles.

Transglutaminase activity is important for skin and is found in both mammals and birds.

GDNF helps grow hair and heal skin wounds by acting on hair stem cells.

Understanding hair follicle development can help treat hair loss, skin regeneration, and certain skin cancers.

A new mutation in the Connexin 26 gene was found in a patient with KID syndrome, expanding the known disorders linked to this gene.