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The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

CDP is crucial for lung and hair follicle cell development.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

Androgens can both stimulate and cause hair loss, and understanding their effects is key to treating hair disorders.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

A certain smell receptor in hair follicles can affect hair growth when activated by a synthetic sandalwood scent.

Lymphatic vessels are essential for hair follicle growth and skin regeneration.

Genetic research has advanced our understanding of skin diseases, but complex conditions require an integrative approach for deeper insight.

Genomic approach finds new possible treatments for hair loss.

Understanding and manipulating epigenetic changes can potentially lead to human organ regeneration therapies, but more research is needed to improve these methods and minimize risks.

Diagnosing sex development disorders requires combining medical history, physical exams, imaging, lab tests, and genetic data.

The hypothesis suggests that PCOS may start early in life due to genetic and environmental factors, influencing future reproductive and metabolic problems.

Male hormones and their receptors play a key role in hair loss and skin health, with potential new treatments being explored.

Genetics play a role in acne, but how exactly they contribute is not fully understood.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

The VEGF +405G allele may increase the risk of PCOS in South Indian women.

The document concludes that PCOS has a strong genetic component, but more research is needed to fully understand the specific genes involved.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Longer CAG and GGN repeats increase alopecia risk, but no significant link to post-finasteride syndrome found.

Wnt proteins from certain skin cells are crucial for normal hair growth and renewal.

Epigenetic mechanisms control skin development by regulating gene expression.

Acne is caused by genetics, diet, hormones, and bacteria, with treatments not yet curative.

The conclusion suggests that prostate cancer should be classified by castration status and that new therapies targeting androgen receptor signaling show promise.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Higher miR-34a levels and the A variant of the MIR-34A gene are linked to increased risk and severity of alopecia areata.

Researchers found 19 genes important for root hair growth in a plant called Arabidopsis.

High levels of TNF-α may contribute to obesity and insulin resistance in PCOS, but not due to the C850T genetic variation.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.