Search

everythinglearnresearchcommunity

for

Search:↓

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Vitamin D deficiency is common in women with PCOS and linked to some metabolic problems, but not the main cause of their metabolic issues.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Minoxidil improves blood flow and vessel flexibility, potentially helping with vascular stiffness.

Vitamin D receptor helps control hair growth genes in skin cells.

New gene mutations linked to skin conditions were found, bacteria and chemicals may worsen acne, a dog mutation could exist in humans, virus-like elements might be involved in psoriasis, and a vitamin D3 treatment doesn't prevent chemotherapy-related hair loss.

A woman with a unique syndrome similar to TRPS has a genetic change near the TRPS1 gene, affecting its regulation.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

The conclusion is that we need more effective hair loss treatments than the current ones, and these could include new drugs, gene and stem cell therapy, hormones, and scalp cooling, but they all need thorough safety testing.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

Using special RNA to target a mutant gene fixed hair problems in mice.

A new mutation in the DCAF17 gene was found to cause Woodhouse-Sakati syndrome in a large family.

Ring Chromosome 11 may be linked to conditions like early puberty, excessive hair growth, hair loss, and type 2 diabetes.

5% topical minoxidil improves hair density and quality in monilethrix patients.

Chronic graft-versus-host disease in the skin shows strong Th1 immune response and unique barrier issues.

A woman with CHILD syndrome showed skin abnormalities, and the report suggests CHILD nevus and NEVIL might be the same condition, highlighting the need for diagnosis for genetic advice.

ANp63 is crucial for skin integrity, new filaggrin gene mutations link to eczema, hair can regrow from non-stem cells, sunburns are increasing, and glucocorticoids help treat skin allergies by affecting immune cells.

Mutations in the androgen receptor gene cause different levels of androgen insensitivity, making it hard to create simple tests for the condition.

Hair pattern in androgenetic alopecia overlaps with scalp and bone demarcations, with distinct gene profiles affecting susceptibility.

Msx and Dlx genes are crucial for development, controlling cell behaviors like growth and differentiation through their roles as gene regulators.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

TTD hair brittleness is caused by multiple structural abnormalities.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Mutations in the androgen receptor gene cause Androgen Insensitivity Syndrome, affecting sexual development.

Keratosis pilaris is often linked to genetic mutations and causes skin and hair abnormalities, regardless of those mutations.

Certain types of mucopolysaccharidoses cause significant hair abnormalities.

The study suggests certain ACE gene variations are more common in women with PCOS and may be linked to increased insulin resistance.

A specific gene mutation causes Olmsted syndrome.

Finasteride exposure affects gene expression and anogenital distance in male rat fetuses.