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Frontal fibrosing alopecia shows increased inflammation and JAK-STAT pathway activity without reduced hair proteins.

Early diagnosis and aggressive treatment are crucial for Cicatricial Alopecia, and treatment effectiveness varies among patients.

Certain immune-related proteins are higher in people with alopecia and their healthy relatives, hinting at a genetic link.

Certain proteins are found at higher levels in balding areas compared to non-balding areas, suggesting a link to hair loss. This could be useful for diagnosing and treating hair loss.

Fetal skin has unique immune cells different from adult skin.

Targeting specific T cells may help treat alopecia areata.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

Piperine from black pepper can make hair less oily by blocking fat cell development in hair roots.

Blocking the protein CXCL12 with a specific antibody can increase hair growth in common hair loss conditions.

Blocking TYK2 might be a new way to treat hair loss from alopecia areata.

The scalp fat tissue of men with hair loss shows changes in gene activity that may contribute to their condition.

A specific RNA molecule blocks hair growth by affecting a protein related to hair loss conditions.

Some multiple sclerosis treatments may trigger hair loss conditions like alopecia areata.

The RNA AL136131.3 slows down hair growth and speeds up hair loss by affecting sugar breakdown in hair follicles.

MPZL3 protein helps control the size of oil glands and the growth of oil-producing cells in both mice and humans.

Booster shots of mRNA vaccines for COVID-19 increased protective antibodies without worsening autoimmune skin conditions in patients.

JAK inhibitors help hair growth by boosting beta-catenin activity in hair root cells.

Type 1/17 inflammation in psoriasis increases skin cell growth due to a molecule that could be a new treatment target.

Lichen planus is a chronic autoimmune disease that is hard to treat and more common in women.

Different types of PPARγ are found in varying amounts in human skin and its parts, which could affect how skin treatments work.

ILC1-like cells may contribute to hair loss in alopecia areata.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Tofacitinib regrew hair in a man with total hair loss but raised cytokine levels, needing more research on possible side effects.

The new aptamer TAGX-0003 shows promise as an effective treatment for hair loss disorder alopecia areata.

TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

Not having the gene PLAAT3 leads to fat loss, high insulin resistance, and abnormal fat levels in the blood due to a disruption in fat cell development and function.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

PPARγ signaling modulation can protect hair follicle stem cells from chemotherapy-induced damage.

Innate lymphoid cells type 1 may contribute to alopecia areata.